Bahman Yousefi, Masoud Darabi*, Behzad Baradaran, Mahmoud Shekari Khaniani, Mohammad Rahbani, Maryam Darabi, Shabnam Fayezi, Amir Mehdizadeh, Negar Saliani, Maghsod Shaaker
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Yalda RahbarSaadat, Nazli Saeidi, Sepideh Zununi Vahed, Abolfazl Barzegari*, Jaleh Barar 
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Abbas Asoudeh-Fard, Abolfazl Barzegari, Alireza Dehnad, Sepideh Bastani, Asal Golchin, Yadollah Omidi*
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Majid Gholipour* , Arezoo Tabrizi
Increased MAP4K1 expression did not influence Lats1 activation. The exercise training protocol suppressed Mst1 and Lats1 (Hippo pathway) and caused an increase in Yap1 expression level, which led to physiological cardiac hypertrophy in healthy rats.
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Aynur Karadağ Gürel* , Selçuk Gürel
As a result, it suggests that the enriched genes and new pathways emerging as a result of the analysis may help identify the group that responds poorly to treatment and the outcome of the treatment.
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Sanya Haiaty , Mohammad-Reza Rashidi, Maryam Akbarzadeh, Ahad Bazmany, Mostafa Mostafazadeh, Saba Nikanfar, Roya Shabkhizan, Rostam Rezaeian, Reza Rahbarghazi* , Mohammad Nouri
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Yuri Yu. Shchegolev , Danila V. Sorokin, Alexander M. Scherbakov* , Olga E. Andreeva , Diana I. Salnikova , Ekaterina I. Mikhaevich , Margarita V. Gudkova , Mikhail A. Krasil’nikov
Prolonged cell treatment with mTOR inhibitor rapamycin leads to the development of resistance. Exosomes of resistant cells induce the resistance to rapamycin in the parent MCF-7cells. Exosome-induced resistance reproduces the changes revealed in the rapamycin-resistant cells, including the activation of ERK/AP-1.
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Farzaneh Bilan , Mohammad Amini, Mohammad Amin Doustvandi, Maryam Tohidast, Amir Baghbanzadeh, Seyed Samad Hosseini , Ahad Mokhtarzadeh* , Behzad Baradaran*
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Naeimeh Akbari-Gharalari , Sina Khodakarimi, Farshad Nezhadshahmohammad, Mohammad Karimipour, Abbas Ebrahimi-Kalan* , Jiagian Wu
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