Yalda RahbarSaadat, Nazli Saeidi, Sepideh Zununi Vahed, Abolfazl Barzegari*, Jaleh Barar 
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Balal Khalilzadeh, Nasrin Shadjou, Hadi Afsharan, Morteza Eskandani, Hojjatollah Nozad Charoudeh, Mohammad-Reza Rashidi*
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Mahdi Edalati Fathabad, Morteza Karimipoor*, Shaban Alizadeh*, Asghar Abdoli, Amir Atashi, Mahtab Sayadi
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Ali Salmassi*, Bengi Acar-Perk, Andreas G. Schmutzler, Kerstin Koch, Frank Püngel, Walter Jonat, Liselotte Mettler
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Hassan Moghtaderi, Houri Sepehri*, Ladan Delphi, Farnoosh Attari
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Hadi Maleki-Kakelar , Jaber Dehghani, Abolfazl Barzegari, Jaleh Barar , Masoud Shirmohamadi, Javid Sadeghi, Yadollah Omidi*
Based on in vitro experiments, the anti-cancer effect of the locally isolated L. plantarum was validated through some signaling pathways, including PTEN, Bax, TLR4, and AKT genes.
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Najme Dehghani , Farzaneh Tafvizi* , Parvaneh Jafari
The supernatant of probiotic L. rhamnosus has a great potential to inhibit the proliferation of HT-29 cells and the induction of apoptosis.
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Sanaz Javanmardi , Samira Sadat Abolmaali* , Mohammad Javad Mehrabanpour, Mahmoud Reza Aghamaali, Ali Mohammad Tamaddon
miR-21 overexpression can result in down-regulation of the critical inhibitory proteins in tumor cells. Anti-miR-21 delivery to resistant ovary tumor cells using the carboxymethylated PEG5k-polyethylenemine nanohydrogels bearing redox-sensitive crosslinks, knockdown the tumor associated angiogenesis and provoke apoptosis though ROS generation.
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Soheila Rostami , Farzaneh Tafvizi* , Hamid Reza Kheiri Manjili
The findings suggest that F-Lys-TMX NPs are more effective and have the potential for cell proliferation inhibition and apoptosis induction compared to the TMX. Hence, F-Lys-TMX NPs can be considered as an anti-cancer agent against MCF-7 breast cancer cells.
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Raghdah S. Bawadud , Mayson H. Alkhatib*
Co-delivery of NE-DTX+TQ may probably inhibit the proliferation of T47D via the induction of apoptosis and autophagy pathways and impede the migration by reducing the BCSC population and downregulating TWIST-1 expression to decrease the epithelial-to-mesenchymal transition (EMT) of breast cancer cells
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Roghiyeh Pashaei-Asl , Maryam Pashaiasl, Esmaeil Ebrahimie, Maryam Lale Ataei, Maliheh Paknejad*
The application of hAFMSCs-CM, as a therapeutic reagent, can suppress breast cancer cells’ viabilities and induce apoptosis.
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Zeinab Mazloumi , Ali Rafat, Khadijeh Dizaji Asl, Mohammad Karimipour, Dariush Shanehbandi, Mehdi Talebi, Majid Montazer, Ali Akbar Movassaghpour, Alireza Dehnad, Raheleh Farahzadi, Hojjatollah Nozad Charoudeh*
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Megha Gautam , Reema Gabrani*
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Sheyda Jodeiry Zaer , Mahmoudreza Aghamaali*, Mohammad Amini, Mohammad Amin Doustvandi, Seyed Samad Hosseini , Behzad Baradaran, Souzan Najafi, Yalda Baghay Esfandyari, Ahad Mokhtarzadeh*
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